Once you have fixed all of these you will be clinically non diabetic. You will pass any diabetic test. Any peripheral neuralgia will have improved to the point of almost vanishing. However you will have to continue to eat fairly low carb and you will have to do some amount of carb burning medium intensity exercise every night before bed for several years after the fix.
The writer was diagnosed Type 2 with fasting sugar of 18 mmol/L (324 mg/dl) in November2012 and HbA1c of 11.4%. He has done all 3 steps above. He now has normal fasting insulin resistance (HOMA IR1 is 1.2), normal HbA1c (4.8%) and normal fasting sugar 4.4 mmol/L.
It was an extremely difficult piece of research to do to get to this point and the writer although clinically non diabetic is not fully cured and still has to watch the carbs and to walk for 75 minutes on a treadmill every night before bed. But his peripheral neuralgia has essentially gone and he has no diabetic complications.
1. Eat two meals per day and no snacking at all. No cups of tea or coffee
except with meals.
2. Eat no type of food with more than 12 grams of carb per 100 grams of food.
3. No Vegetable oil at all
4. No Nuts at all.
5. No Cheese at all
6. No sugar at all..
7. 300-400 grams of flax seed and chia seed bread per day
8. 100-200 grams of full fat natural yoghurt per day
9. 250-400 grams of whole milk per day
10. Fish or Chicken or Lamb or Beef roasted or grilled so as to remove most of the fat.
11. Eat at most one egg per day.
12. Eat spreadable butter which is mainly butter with a small amount of rapeseed oil (such as Lurpak spreadable)
13. No bread, no grains, no cakes, no biscuits, no pizza, no pastries, no pasta, no muffins, no doughnuts, no crackers, no pies
14. No fruits except a handful of raspberries, blackberries, strawberries of an eight of a grapefruit
15. Exercise upon a treadmill or a spin bike after your last meal for between 45 and 150 minutes until your sugar is less than 5.0 mmol/L (90 mg/dl). Take as many 3 minute breaks during the exercise as you need. Fast walking.
16. No processed meat (smoked, salted, cured or preserved) or aged meat. No pork pies, sausages, bacon, cured hams, luncheon meat, etc. Preserved meat causes more gluconeogenesis - your body will just melt it down for sugar.
17. Non fish seafood such as prawns, crab, lobster squid causes excessive gluconeogenesis too and should be limited or avoided.
18. Plenty of green vegetables (not avocado - too much mono and not kale).
19 Take all the following daily supplements and no others (for diabetes).
4 grams EPA from molecularly distilled triple strength fish oil (I use 8x
Holland and Barrett 1360 mg and 5x Dr's Best Calamari DHA 500)
4 grams DHA from molecularly distilled triple strength fish oil (I use 8x Holland and Barrett 1360 mg and 5x Dr's Best Calamari DHA 500)
5000 IU Vitamin D3
25,000 IU Natural Beta Carotene
10,000 IU Vitamin A from fish oil
500 mg Vitamin C
600 mg Meriva Curcumin (I use X4000)
500 mg Magnesium (I use Life Extension)
2 heaped teaspoons of good quality fragrant not dry true cinnamon (from Sri Lanka) not cassia cinnamon, with some whole milk in a hot drink. Cassia thins the blood and so a bad idea.
0. Abbot Freestyle Libre Continuous Glucose Monitoring system (or other CGM
system). You cannot fight something that you cannot see.
1. Access to a treadmill
2, Kenwood chef
3. Nutribullet blender
You will need to bake our flax seed and chia seed bread - it is really easy. On the first day I would eat nothing all day until dinner and then have the following
Take all the supplements with 300 ml of whole milk.
200 grams of natural yoghurt. Eat this first to line the stomach and help the good bacteria fight the bad ones.
4 thick slices of flax seed and chia seed bread. 2 with spreadable butter and 2 with salmon spread or tomato salsa or pureed vegetable spread or lumpfish roe.
Large portion of roast chicken and boiled turnips or swede with spinach or broccoli. Roast the fat out and pour the fat out to make the gravy.
Then walk upon the treadmill as a brisk pace stretching yourself but not stressing yourself, just be at the point where you have to start breathing through your mouth rather than your nose - this will be 50% of VO2 max. You will probably have to walk for 3 hours on the first day to get your sugar numbers to be half decent. Get the most impact absorbent trainers you can and use impact absorbing insoles inside then. I use 2mm Noenes in addition to Sorbothane pro insoles, PROTECT YOUR FEET. If your feet get blisters or sores they will heal very slowly due to type2. Your feet are your main weapon against type2. And it is as if type 2 knows this. It will mercilessly attack your feet.
This is a low carb, low vegetable fat, low animal fat, high non cheese milk fat, high Omega3 fat diet.
We add to this a carb burning medium intensity exercise regime (after all meals at the end of the day). This will burn more carb grams than you have eaten. That fixes sugar levels. So the combined treatment is...
A Carb Negative Insulin Resistance Reducing Fat Only Diet and Exercise Regime. a CNIRRFODER.
Carbs raise blood sugar. Omega6 and Omega9 vegetable fats raise insulin resistance. Omega3 and Omega7 fats do not raise insulin resistance, they improve it. But Omega7 fats are only found naturally together with too much Omega9. Macadamia nuts are the purest natural source of Omega7. That is why if you are going to eat any nuts then Macadamias are the best (having almost no Omega6 and a lot of Omega7). But even Macadamias do cause insulin resistance due to their Omega9 mono fat content. Omega3 is only found in sufficient quantity to be beneficial in flax seed and chia seed. Only eat WHITE chia seed (salba seed). Do not eat BLACK chia seed (less carefully regulated than salba - causes higher sugar). .
For the full nutritional breakdown see diabetic-index.html
It should take you a week of long hours upon the treadmill every night to get your spot sugar down to 5 mmol/L or 90 mg/dl.
Initially you may have to walk for 2-3 hours each night. It takes a while to clear out all the excess sugar from all over your body. Eventually things will settle down at between 40 mins and 90 mins per night for perfect post exercise sugar depnding upon how badly diabetic you have been.
And that is the fix. We do not go low carb high fat. We go low carb low Omega6/9 fat, low animal fat, high non cheese milk fat (whole milk, butter and full fat yoghurt), high short and long chain Omega3 fat. And we take anti inflammatory supplements which block delta 5 desaturase and fix the imbalance in our Omega6 metabolism caused by excess insulin which linearly up-regulates our delta 5 desaturase levels and therefore pushes our Omega6 metabolism to be inflammatory.
Get a continuous Glucose Monitoring device (CGM). The Freestyle Libre will be around 20% out on day 1 and 10% out on day 2. But it is pretty good from day 3-14 (within 5%). Each patch only works for 14 days. You should be able to fix your type 2 within 2 patches.
Continue as for Step1, but for a month.
Continue as for Step1, but for 3 months. You will find that your walking time reduces down to a residual level and if you go to the doctor and measure your insulin and sugar after having eaten nothing for 12 hours you will get your fasting insulin resistance from the formula...
HOMA IR1 = fasting insulin (mIU/L) x fasting sugar (mmol/L) /22.5 = 1.1 for a normal person and is above 1.8 for a type 2 diabetic.
Mine is 1.2. I can fix fasting sugar, HbA1c and Insulin Resistance. But I cannot get the walking time to zero in my case. I suspect that Type 2 Diabetes is caused by an intestinal parasite bacteria which somehow hacks your sugar system to turn your body in to a sugar factory for the parasite. It is not merely caused by Insulin Resistance.
Prior to diagnosis. 2/3 bottles of lucozade per day, 3 cans of coca cola per day, 4/5 pieces of coffee shop cake, and a snickers bar every day!!
Diagnosis Day1 (2014June10): Spot sugar 13.9 mmol/l, HbA1c 10.6% (92 mmol/mol). He had been fully diabetic for 2 months, normal weight, sedentary high stress fast food high carb lifestyle (London minicab company owner and driver 32 years old). His doctor told him he must go on Metformin immediately. He declined. Height 5ft 8½ inches, 70 Kg. BMI 23.
Day2: He called me and I said walk for 20 minutes after every meal and stop eating sugar and carbs.
Day5 (2014June15): He came to see me after having partially followed my instructions - his spot sugar was now 10.3 mmol/l (185 mg/dl). I told him to get on the treadmill. He walked for 30 minutes at 5.6 kph. Then his sugar was 7.1 mmol/l (128mg/dl). This astonished me because if I walk for 30 mins at 5.6 kph my sugar will go down by 1.5 mmol/l not 3.2. So then I said to hell with it, let's finish this, and he walked another 30 minutes at 5.6 kph for the first half and at 5.8 kph for the second half. This took his sugar down to 4.9 mmol/l (88 mg/dl). That astonished both of us. We had got his sugar to normal in one day. I then told him he must walk for 30 minutes after every meal on the treadmill at his local gym and he must eat a very low carb diet, nothing with more than 10% carbs in it.
Day11: He followed some of my instructions and walked for 30 minutes after every meal on the pavement not on the treadmill. He was still eating porridge for breakfast. His sugar was normally around 6.2 when he got up and around 6.2 after each walk. I told him he must walk upon the treadmill not upon the pavement.
Day18: Having walked upon the treadmill as instructed although still eating some porridge and drinking fully caffeinated coffee and having the odd wholemeal bread starbucks sandwich his sugar was now around 5.5 mmol/l (99 mg/dl) in the morning and after every walk. So now his diabetic symptoms have gone and he is clinically non diabetic as of June29. We will carry on with this program for another week and see what happens next.
Day 30 (2014July15): Walking 30 minutes on the treadmill each day and a further 20 mins after dinner outside, for a total of 50 minutes per day, and eating a 100 carb gram per day diet (still eating porridge for breakfast - bad idea!) he now has a morning sugar of 5.2-5.5 mmol/L (94-99). So that is it. He is clinically cured and can maintain the cure on less than an hour's exercise per day with a 100 carb gram per day diet. He got there in 30 days.
Day 36 (2014July21): Starting a new regime for 4 weeks of walking 45 minutes on the treadmill once per day. Breakfast is porridge + milk in morning with a quarter of a grapefruit, and vegetable juice. Lunch Salmon or Sardines or Cod or Sea Bass + Salad, Green Tea or Mint Tea. Dinner: Peanut butter on low carb bread and salad and soup, or lamb chops and salad (kidney beans, cheese, cucumber, avocado, lettuce, cabbage, tomato, olive oil, vinegar), glass of semi skimmed milk. This is a weight maintaining diet.
Day 40 (2014July25): Regime is going fine. His morning sugar is still between 5.2 and 5.5 mmol/L (94-99 mg/dl) walking once for 45 minutes each day on the treadmill and taking a 100 carb gram per day diet.
Day 122 (2014October16). Sami had trouble with his feet ( I think the treadmill at his health club did not have good enough suspension). So his diet was OK but he has been unable for the last month to do much walking. Anyway his HbA1c results came in today and he was 40 mmol/mol or 5.8%. That is non diabetic. He is clinically cured. 5.8% is at the high end of the non diabetic range. 5.5% or below is the target in the writer's opinion. But anything at or below 6.0% is a clinical cure. So he is cured. He managed this by restricting carbs to 100 grams per day and by walking for 45 minutes per day on a treadmill for 3 months and could not manage any walking in the 4th month. He stopped exercising in the last 30 days due to pain in his feet. So there you are, a second type2 cure in 4 months with a limited amount of carb burning exercise and a low carb but not an ultra low carb diet. So it can be done relatively effortlessly if you catch type2 early enough.
His doctor (having recommended drugs and even insulin) was amazed and asked him how he did it. He said low carb diet and exercise.
Joe had a fasting sugar of 9.0 in January 2014 and an HbA1c of 7.4%. He was mildly diabetic on diagnosis. He weighed 82 kg at the time. He made a decision to adopt the 5:2 intermittent fasting diet of Doctor Moseley, rather than a full Newcastle reversal scheme of Prof Taylor. He combined the diet with low carb and with HIT training every day. The results are a text book example of how to reverse diabetes.
Low Carb (50-60 grams per day)
4 days of 12-15 min HIT per week
3 days of 40 minute weight lifting sessions per week
5:2 Intermittent fasting diet.
Plenty of Low Carb Australian Beer!
|Date||Fasting Sugar mmol/L||Weight Kg||Blood Pressure mm hg|
|Week5||5.6||76||131 (sick - no HIT)|
WOW! - Gordon. Joe only had mild diabetes. But he killed it completely with HIT, Low Carb and 5:2 fasting and Australian low carb beer in 9 weeks! (I have seen copies of his lab results). He used a Low Carb HIT 5:2 Newcastle reversal. Or putting it another way, he used a Carb Zero 5:2 Newcastle reversal. Very effective - so long as you are 18% above normal BMI to begin with.
There is absolutely no drug that can do that!
I recommend moderate intensity exercise (brisk walking upon a treadmill). But Joe's results indicate that high intensity works just as well (as do the results of Mark Quade - see Testimonials). So at the end of the day you just have to burn off the carb grams that you eat it appears - one way or another (with moderate or high intensity exercise).
Vikas has the most astonishing story that I could hardly believe before I saw his lab results.
Vikas was diagnosed on 15th Jan 2015. He was continuously so tired that he
could not even walk 100 meters. His fasting sugar was 200 mg/dl and his post
prandial sugar was typically 265 mg/dl. His HbA1c was 10.4%. His Vitamin D was
only 12 (should be between 75 and 200). His doctor instructed him to start taking
insulin immediately and he refused. Instead he took 3 diabetes tablets daily (Voglibose,
Metformin and Gemer P1 - which is a combination of Pioglitazone, Glimepiride and
Then he read this website and decided to go for it. His regime was 1 hour of brisk walking every day in hard sunlight after lunch and a 1 hour intensive workout in the gym along with the 3 tablets and a hell of a lot of Vitamin D (around 10,000 IU per day!) Then after a mere 10 days on this regime his HbA1c came down to 8.7%. He continued this regime for a further 20 days and on 15th Feb his HbA1c was 5.1%, his fasting sugar was 96 mg/dl and his post prandial sugar was 103 mg/dl. His doctor then reduced his diabetic medication to one tablet. So if this is to be believed then he reduced his HbA1c from 10.4% to 5.1% in 30 days. The reason that I have included Vikas' figures on the home page of this site is that first of all he sent me all 3 of his blood test results for Jan15, Jan26 and Feb15, and secondly I have now realised that HbA1c is not a 3 month blood sugar average, but is actually a 4 week blood sugar average weighted toward the most recent 2 weeks. This is very well explained on the American Diabetes Association website at...
"Hb = hemoglobin, the compound in the red blood cells that transports oxygen. Hemoglobin occurs in several variants; the one which composes about 90% of the total is known as hemoglobin A. A1c is a specific subtype of hemoglobin A. The 1 is actually a subscript to the A, and the c is a subscript to the 1. "Hemoglobin" is also spelled "haemoglobin", depending on your geographic allegiance.
Glucose binds slowly to hemoglobin A, forming the A1c subtype. The reverse reaction, or decomposition, proceeds relatively slowly, so any buildup persists for roughly 4 weeks. Because of the reverse reaction, the actual HbA1c level is strongly weighted toward the present. Some of the HbA1c is also removed when erythrocytes (red blood cells) are recycled after their normal lifetime of about 90-120 days. These factors combine so that the HbA1c level represents the average blood glucose level of approximately the past 4 weeks, strongly weighted toward the most recent 2 weeks. It is almost entirely insensitive to blood glucose levels more than 4 weeks previous.
In non-diabetic persons, the formation, decomposition and destruction of HbA1c reach a steady state with about 3.0% to 6.5% of the hemoglobin being the A1c subtype. Most diabetic individuals have a higher average bG level than non-diabetics, resulting in a higher HbA1c level. The actual HbA1c level can be used as an indicator of the average recent blood glucose level. This in turn indicates the possible level of glycation damage to tissues, and thus of diabetic complications, if continued for years."
With this new understanding of the true meaning of HbA1c, Vikas' figures are completely credible. So if you throw the kitchen sink at type 2. If you take the medication and if you go zero or negative carb by walking off all the carb grams you ingest and if you add some high intensity training 3x a week, then you may, like Vikas, be able to kill it dead in 30 days!
Do not do High Intensity Training on the same muscle group every day. Do it every other day at most or it becomes counter productive. The muscles need to recover and rebuild themselves during the day after each session.
I was diagnosed on November28, 2012, with a spot sugar of at 23 mmol/l (414 mg/dl), a fasting sugar of 18.0 mmol/L (324 mg/dl) and an HbA1c of 11.4% and BMI of 26.7. I had been extra thirsty for two years and had had neuralgia in the 4th toe of my right foot for 12 months - which I mistook for an in growing toenail. So I was very badly diabetic type 2. I had plenty of insulin but no glycogen storage space in my muscles and I had significant insulin resistance (I still have no idea what the real cause of this is. It is initiallly caused by high sugar and high insulin. But reducing them both back to normal for 2 years in my case has not completely fixed it - although it has improved it. Insulin resistance is not caused by microvascular capillary atherosclerosis caused by AGEs - Advanced Glycation End products - because some severe diabetics just do not get this and therefore have no peripheral neuralgia).
Here is a letter from my Doctor confirming my journey as of July 20, 2015...
19 December 2013
Mr Gordon Ritchie
By Email: email@example.com
was a pleasure to catch up with you again the other day.
I thought the research you have performed in relation to diabetes and
muscle mass was very interesting. You
have successfully cured the diabetes which was identified in November 2012 by
a combination of diet and exercise. Originally
the random blood glucose was 18 with a haemoglobin A1c of 11.4% (101 mmol/mol).
You told me that you had also been found to be deficient in vitamin D
and had been taking a supplement, although at a relatively low dose by the
sounds of things.
am pleased to inform you that your haemoglobin A1c is now 5.3% (35 mmol/mol).
This is well within the normal limits.
Your lipid profile is favourable with regards to future risks of heart
disease with a total cholesterol of 3.9, 31% healthy HDL (absolute value 1.2),
leaving an LDL of 2.2. The only
fly in the ointment is the vitamin D level is still low at 36.
You know that you need to keep it up.
I think you need a significantly increased dose of vitamin D and would
suggest for a month that you take 2000 international units daily (available
over the counter) before reducing the dose back to 1000 international units
Christmas and happy New Year.
XXXXXXXX MRCP (UK) DFOM
reference number 2709985
Here are my latest blood test results showing my HbA1c
on 2015July20 at 5.1% or 32 mmol/mol and my Vitamin D at 143 nmol/L. I had been
taking 5,000 IU of Vitamin D3 per day for several months.
are my continuous blood sugar results as measured by the Abbott Freestyle Libre
Continuous Glucose Monitoring device for the week of August 21-27, 2017
Here are my continuous blood sugar results as measured by the Abbott Freestyle Libre Continuous Glucose Monitoring device for the week of August 21-27, 2017
The solution is to be very specific about what fats you eat and what fats you do not eat. Do not just eat less carb and more fat. That will definitely put your insulin resistance up. It did with me for 3 years notwithstanding the perfect non diabetic sugar numbers I had due to all the exercise and the low carb diet. The good fats for a diabetic are fish based Omega3 anti-inflammatory fatty acids and plant based Omega3 (alpha linolenic acid) from flax seed and white NOT BLACK chia seed and milk fat in the form of butter whole milk or yoghurt NOT in the form of cheese, and that is it. The bad fats are sat fat from meat (particularly palmitic acid) and Omega9 mono fat (which is a lot less harmful) and Omega6 poly fats which are not a problem in moderation for normal people but are a disaster for type2 diabetics whose increased insulin up-regulates their desaturase enzymes to the point where their Omega6 metabolism ceases to be balanced and becomes inflammatory which causes insulin resistance. Industrial Trans fat is of course also a total disaster for everybody, whereas ruminant trans fat such as in meat and dairy is perhaps only slightly worse than sat fat.
It does not seem to matter what type of Omega6 you eat. It all manages to end up as Arachidonic Acid which is the most inflammatory fat there is.
If high insulin resistance persists for months then capillary plaque may also start to form. But the writer has had type for around 10 years (6 of which were undiagnosed) and even now his non fasting insulin resistance appears to be more related to inflammation than plaque (cholesterol is fine). Inflammation has a larger effect upon capillaries than it does upon larger vessels. If you thicken up the wall of the small capillary it will seriously restrict its already small diameter. Whereas if you thicken up the wall of a larger vessel the internal diameter of that vessel will not change so drastically. The inflammatory part of that response is multiplied by the western diet which is far too rich in pro inflammatory Omega6 fats and far to poor in anti inflammatory Omega3 fats (typically around 20:1 when it should be 1:1 for a normal person and 1:2 for a type 2 diabetic and 1:4 up as far as 1:7 for an eskimo).
The modern day American has a blood Omega6:Omega3 ratio of around 10:1. The traditional Eskimo has 1:7 the other way around. Eskimos on their traditional diet get neither type 2 diabetes nor coronary heart disease.
Technically: Omega6 is mainly present in food as linoleic acid (18:2ω6) which is converted by the body to AA (Arachidonic Acid, 20:4ω6). Whereas Omega3 is mainly present as alpha linolenic acid (18:3ω3) which is converted by the body to EPA (20:5ω3). The ratio of AA to EPA in your blood determines your inflammatory response. This is easy to fix. Mine as of 2016November is 0.78, which is around the level of a traditional Japanese fish diet consumer. The healthy male body only converts around 8% of the ALA available to EPA. The healthy female body converts around 20%. The same figures apply to conversion of LA to DGLA and to AA in the Omega6 pathway. So in theory one gram of EPA will defray 12½ grams of Omega linoleic acid from food in men and 5 grams in women. I have not found that to be true in practice. This is because I am type 2 diabetic and therefore have higher insulin levels and therefore have higher desaturase levels and the desaturases are the rate limiting enzymes in both the Omega3 and the Omega6 pathways. I would say I gram of EPA protects against perhaps 5 grams of LA for a type 2 male diabetic like me. For a female it will be worse since they have more desaturases in the first place.
So by taking 5 grams of EPA per day (and 5 grams of DHA to balance it), one is actually only protected against around 10 grams of Linoleic Acid (LA) if male (in my experience). It will be worse if female (since women have higher desaturase levels). So that is how dangerous Omega6 fat is to a type 2 diabetic. Hence having fish on Friday and cod liver oil every day is not such a bad idea. Because type2 diabetics have higher insulin levels and so have higher levels of the enzymes which catalyse the synthesis of EPA and AA, males may convert 16%/24% or more of LA to AA and type2 diabetic women up to 50%. It all depends upon how high your insulin is, and that depends on how bad your insulin resistance is and that is the true measure of how diabetic you are.
So we need to go Eskimo, since they eat 4x-7x as much Omega3 as they do Omega6 on their traditional seafood diet. Eskimos on their traditional diet get neither Coronary Heart Disease nor type 2. Whereas westerners eat more than 20x more Omega6 than Omega3 on their traditional fast food diet. But taking one or two or three regular strength or even triple strength Omega3 pills per day will absolutely not fix type 2. Eskimos eat around 15 grams total of EPA and DHA per day with little or no Omega6. And on that diet no Eskimo ever became diabetic because it is entirely anti inflammatory. If you have type 2, then your muscle cell membranes will be made up of the wrong fats and so are your blood cell membranes.
For a fairly full explanation of the cause of Insulin Resistance please see our insulin resistance page.
For the full breakdown of the inflammatory or anti inflammatory effect of 16,417 foods taken from the US, the UK, the Australian and the Norwegian national databases please see our Diabetic Index Table.
Type 2 diabetes, metabolic syndrome and high blood sugar are caused by a failure of your body to store post prandial sugar as glycogen in your liver and in your muscles - due to insulin resistance cause by inflammation and by excessive glucagon mediated hepatic gluconeogenesis - that is to say an excessive overproduction in the liver of sugar from excess protein that you have eaten. The liver will stop overproducing if you get the diet and exercise routine right (mine no longer does).
In lay man's terms, either your sugar warehouse is too small due to muscular degeneration from a sedentary high carb lifestyle, or your muscle cell insulin receptors are gummed up with Omega6, which is preventing insulin from doing its job in your muscle cells or your fork lift truck is driver inside your muscle cell is unable to do much work because the whole warehouse if full of toxic waste products (reactive oxygen species) resulting from metabolic overload or the roads supplying your sugar warehouse are blocked due to vascular sclerosis. You have a logistical problem with sugar. But glucose intolerance is not the cause of Type2. It is a symptom of the disease.
With Type2 your body gets caught in a feedback loop. Higher sugar leads to all of the above causes getting worse which leads to even higher sugar. Then after a few months of this disaster, secondary complications begin. These include retinal blindness, kidney failure, neuralgia (pins and needles in your nerves due to poor blood supply to them), poor peripheral circulation (in the feet and hands) - which if left untreated can lead to amputation, lower skin resistance to infection and slow skin healing from cuts and bruises, skin bacterial infections such as erythrasma, and skin fungal infections such are ringworm and thrush. Then as the vascular sclerosis progresses from the smaller vessels in your skin to the larger vessels in your body, you can get heart attacks and strokes which can kill you. Incidentally type 2 causes impotence in the majority of men since potence requires good blood circulation and sensitive nerves in that area.
That is the bad news.
Now here is the good news.
You can absolutely stop Type2 progressing by going on a carb negative diet and exercise regime. This is conceptually quite simple to grasp but requires effort to implement in your life.
The way to lose weight is to burn more
calories with exercise then you eat in a day.
The way to lower blood sugar is to burn more carb grams that you eat and can store in a day.
A non diabetic person who eats 100 grams of carbohydrate will store the resulting glucose as glycogen in his muscles and in his liver within 2 the hours following his meal and then use that stored energy keep his muscles and body going until the next meal. A Type2 diabetic cannot do that. So what we have to do is burn the glucose that we cannot store with post prandial exercise (exercise after the meal). So instead of storing it, we burn it. It is that simple. And we should burn it at the same time that a non diabetic person would be storing it - i.e. as soon as possible after the meal. I have a treadmill. And after each meal I jump on that and watch something on youtube or an iplayer or Amazon prime or whatever.
If you eat a low carb meal and exercise straight after it then you fix your sugar and diabetes will stop progressing and start regressing.
Diabetes progresses when HbA1c is above 6% and regresses when HbA1c is below 5.5%. Diabetes goes into regression (desugarization) the minute you take your blood sugar below 6.0 mmol/L or 108 mg/dl and it goes into progression (resugarization) the minute you sugar rises to 7.0 mmol/L or 126 mg/dl. Type 2 diabetes cure and reversal can be achieved through Ketosis resulting from a carb negative diet and exercise regime. Any sufficiently carb negative diet and exercise regime will significantly outperform all the following drugs: Metformin, Victoza, Januvia, Glipizide, Lantus, Janumet, Glimepiride, Humalog, Actos, Invokana, Diamicron, Amaryl, Pioglitazone, Onglyza, Gliclazide, Lucentis, Byetta, Galvus, Acarbose, Forxiga, NovoRapid, Exenatide, Apidra, Liraglutide, Repaglinide, Actrapid, Eucreas, Glucobay, Saxagliptin, Vildagliptin, Dapagliflozin, Canagliflozin, Prandin. These drugs can get your HbA1c down to 6.5% at best (which is well managed but still diabetic). A Carb negative diet and exercise regime can get it down to 5% which is non diabetic. With an HbA1c of 5% your diabetes is in full regression. With an HbA1c of 6.5% it is in slow progression.
I have been doing this for 4 years now (as on November 2016) and so have many readers of this site. We have all reversed diabetes in the sense that is no longer progressing and we will not get any further complications. In my case my peripheral neuralgia in my feet has all but gone. I can walk around barefoot with no problem, which is impossible for people with neuralgia. My HbA1c is totally normal at 4.6% which is 26 mmol/mol. My blood sugar is totally normal around 4.4 mmol/L which is 80 mg/dl. My fasting insulin resistance is also normal as of early 2017. But I am still diabetic presumably due to no fasting insulin resistance or in my recent opinion due to having a bacteria in my gut which causes this disease. I think perhaps I am a machine for producing sugar for an intestinal parasite.
When I first got my treadmill in mid 2013, I had to walk 180 minutes per day for imperfect but OK sugar (I was severely diabetic). Then by 2015, I only had to walk for 105 minutes per day for perfect sugar. But in January 2016, I was walking for 140 minutes per day and realised that something was going wrong. I then started treating my insulin resistance as well as my blood sugar and within 4 weeks I only had to walk for 105 minutes per day once more. Now, as of November 2016, I only have to walk for around 75 minutes per day. So I am very nearly there. The immoveable boulder has now been rolled back almost to the beginning - see Insulin Resistance.
Drugs can reduce your HbA1c (Glycosylated Haemoglobin level - 1-2 month blood sugar average) by around 1% in the medium term (according to their clinical trial results). But that is not enough to put diabetes into regression. All the drugs will do is slow down the rate of progression of the disease. To put the disease into regression you need to get your HbA1c down to 5.5%. Generally Diabetics have an HbA1c of anything from 7% to 12%. So you need to reduce your HbA1c not by 1% but by at least 1.5% and normally by 2%-5%. No drug combination can yet do that without lifestyle change as well. So drugs will buy you a bit of time. But they will not cure you unless you are prepared to help them and cure yourself with some lifestyle improvement.
The only way to achieve a type 2 diabetes cure, a full reversal, generally, is by going on a carb negative diet and exercise regime. You must burn off more carbohydrate than you eat - with carb burning exercise. So the cure is to go on a very low carb diet (we recommend 40-50 carb grams per day maximum) - see http://www.nutritionjrnl.com/article/S0899-9007(14)00332-3/fulltext and see http://www.diabetes.co.uk/news/2014/jul/researchers-recommend-carb-restriction-as-primary-method-of-diabetes-control-91152031.html and to go on a brisk walk after every meal (preferably upon a treadmill) to burn off the carbs you have just eaten and the new glucose that your liver has made from the protein you have just eaten by hepatic gluconeogenesis.
The fastest way to fix your sugar is to stay on the drugs initially and change your lifestyle by going Carb Zero or Carb Negative. Then, once your fasting sugar and your HbA1c is fixed, you can start reducing the drugs down to zero in consultation with your doctor. If you stay on any drugs for too long without getting your sugar low enough to put diabetes into regression, they will start to be less and less effective and you have to take more and more of them until your become insulin dependent as your pancreatic beta cells progressively fail. Please do not let that happen. Change your lifestyle. You will feel better, you will be better and you will live much longer.
Here is the golden rule...
Diabetes Progresses when your spot sugar is above 7.0 mmol/L (126 mg/dl),
(HbA1c above 6%)
Diabetes Regresses when your spot sugar is below 6.0 mmol/L (108 mg/dl), (HbA1c below 5.4%)
And here is the philosophy for a diabetic...
Carbs are poison. Exercise is the
Mono fat (Omega9) and Omega6 Poly fat and animal fat are the poisons. Omega3 poly, Omega7 poly and milk fat from whole milk or natural yoghurt but not from cheese are the antidote!
Take the antidote every time you take the poison, and take it as soon as you can after poisoning yourself. Excess Carbohydrate over and above what the brain needs to keep functioning (which is around 75 carb grams per day) is 100% poison. Some fats are poisonous and others are not. Protein is around 50% poisonous due to hepatic gluconeogenesis (the liver will turn around half of it into carbs on a very low carb diet). Type1 diabetics are advised to treat protein like half the amount of carb when calculating how much insulin is required. Do not worry about eating a high fat low carb eskimo diet. Excess carbs cause weight gain and cardio vascular trouble not excess fat - however counter intuitive that may seem or feel. Low fat foods have resulted in an epidemic of fatness. What we need is low carb foods!
This is all is easy to say (although it was not so easy to deduce) - but extremely difficult to do (until you know how). However the writer has done it and he was badly diabetic and not obese at diagnosis on 2012November26 (spot sugar was 23.0 mmol/L 414 mg/dl and HbA1c was 11.4% with a BMI of 26.7). Also a friend of his has now done it completely in just one month following our latest techniques. He was diagnosed on 2014June10 with spot blood sugar of 13.9 mmol/l (250 mg/dl) and HbA1c of 10.6%, and had been extra thirsty for 2 months prior to that - so we assume he had been fully diabetic for 2 months. And many readers of this site have done it and written in with their results - see testimonials.
We must make one thing clear. It does not matter how your reduce your HbA1c down to 5.4%. You can take every drug in the book and eat no carbs and sit on your butt all day long. You can eat a half a chocolate cake every day, take no drugs at all, and go jogging for 2 hours after the cake. You can combine any mixture of drugs exercise and diet you like.
But you MUST get your 2 hour post
prandial sugar down to 6.0 mmol/L, 108 mg/dl (without post prandial exercise)
Then you MUST get your fasting sugar down to 5.2 mmol/L, 94 mg/dl every morning
Then you MUST get your HbA1c down to 5.4% and keep it below there.
Do not become a spectator at you own personal sugargeddon. Take some action!
The writer is not against diabetic drugs. He is just pointing out that they will not, by themselves, stop diabetes killing you. They will not, by themselves, stop you developing painful and debilitating diabetic complications. Do whatever combination of drugs and exercise and low carb dieting works for you. Drug assisted diet and exercise is fine (although certain drugs carry the risk of hypos with high intensity exercise). However for those of you who, like the writer, are wary of the profit driven offerings from the modern pharmaceutical industry, the good news is that diet and exercise are more powerful and more effective than drugs in the case of type 2 diabetes and further, that a sufficiently carb negative diet and exercise regime will in our experience immediately put diabetes into regression without the need for any drugs at all.
The writer cannot over emphasize that it is not in general the case that merely by losing weight you can reverse type 2. A Low Carb Ketogenic diet is for a fact more beneficial than a Low Calorie Diet even if you were obese upon diagnosis - see http://www.nutritionandmetabolism.com/content/5/1/36 and see http://www.nutritionjrnl.com/article/S0899-9007(14)00332-3/fulltext . If you were clinically obese and only slightly diabetic at diagnosis, then losing weight should do it. But type 2 is caused by your body having insulin resistance and insufficient glycogen storage capacity in its skeletal muscles for your typical carbohydrate consumption and some kind of endocrine system fault/cerebral sugar addiction which causes the liver to make too much sugar or not store enough of it as glycogen. It is not caused by being fat. Being fat just increases your metabolic load and so requires your body to have a larger glycogen battery.
If you diet below a BMI of 24 for a man or below a BMI of 22.5 for a woman then you are just eating up your own muscles. But these are critical in storing sugar as glycogen and preventing diabetes from progressing. Sami, a friend of the writer, was fixed in 30 days with no weight loss whatsoever. The writer himself made the mistake of following the Newcastle Reversal concept that if you lose 15% of you body weight you cease to be diabetic. He lost more than that and destroyed his muscles and locked myself in a weak and sick and diabetic state for months. It was a DISASTER. The Newcastle Reversal Technique (low calorie weight loss diet alone) only works on really obese people who were mildly diabetic on diagnosis. Prof Taylor chose people with an average BMI on 33.6 and a fasting sugar of 9.2 mmol/L (166 mg/dl). My BMI was 26.7 on diagnosis and my fasting sugar was 18.0 mmol/L (324 mg/dl) and I dieted down to a BMI of 21.0. I mistakenly thought that there was a weight at which I would become non diabetic. Nothing could be further from the truth. Weight loss only helps if you were very overweight upon diagnosis.
Around 100 grams or 1/6th of your glycogen is stored in the liver and around 500 grams or 5/6th of it is stored in your muscle cells - see http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3248697/. So eliminating all the fat around your liver by losing weight is unlikely to fix the problem as Prof Taylor of Newcastle University suggests. I tried it. I went from 176 lbs to 138 lbs in 14 weeks. This improved my diabetes to the point where I could almost pass a 50% OGTT. But it did not fully reverse my diabetes. In fact it damaged me and increased my neuralgia, by eating away some of my muscle tissue. Also diabetic who have liver transplants from non diabetic donors remain just as diabetic with their new liver. So the liver is not the cause of diabetes. In general to fix type 2 you need to fix not only your diet and your weight but also your muscles and your insulin resistance and your cerebral sugar addiction. In my case I had to reverse 30 years of muscular degeneration caused by a high carb sedentary lifestyle! That does not happen overnight and there is no pill which gives you fitness. Neither can it be achieved by diet alone. As of August 2014 my BMI is now 23.5, which is still a little low given the amount of muscle I have put on from brisk treadmill walking and pull ups.
For mankind's latest understanding of "The Role of Skeletal Muscle Glycogen Breakdown for Regulation of Insulin Sensitivity by Exercise" published in Frontiers in Physiology in December 2011 - see http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3248697/